Cadmore media player playing video Tendon Imbalances: Mallet Finger and Swan Neck Deformity
Video Player
Accessibility
Using forms mode, you can utilize keyboard shortcuts to make usability more efficient.
Space bar will play and pause the video. Pressing escape will close all displays and pressing it again will jump to the end of the player.
You can press A to open the Keyboard Shortcut Dialog which also provides an audio description of the player and all of its features.
Language
1x
Play Speed
Adjust Volume
Transcript
Search
Result Count
of
Click to jump to result
Search
Re-center
Segments
Result Count
of
Click to jump to result
Search
Name:
Tendon Imbalances: Mallet Finger and Swan Neck Deformity
Description:
Tendon Imbalances: Mallet Finger and Swan Neck Deformity
Thumbnail URL:
https://cadmoremediastorage.blob.core.windows.net/a0ed7ec1-23dc-49fa-81ea-58b20b274a9c/videoscrubberimages/Scrubber_1.jpg
Duration:
T01H36M09S
Embed URL:
https://stream.cadmore.media/player/a0ed7ec1-23dc-49fa-81ea-58b20b274a9c
Content URL:
https://cadmoreoriginalmedia.blob.core.windows.net/a0ed7ec1-23dc-49fa-81ea-58b20b274a9c/v-004474.mp4?sv=2019-02-02&sr=c&sig=VyJC3eNU7hZRg2meTatOX5DPi%2BsAKG3jxH%2Bw3aw6M%2Bc%3D&st=2024-11-22T04%3A50%3A38Z&se=2024-11-22T06%3A55%3A38Z&sp=r
Upload Date:
2024-05-31T00:00:00.0000000
Transcript:
Language: EN.
Segment:0 .
JOSEPH M. SHERRILL: Right. Tendon Imbalances. Dr. Joseph M Sherrill. This discussion of tendon imbalances focuses on the diagnosis and treatment of mallet finger and swan neck deformity and includes discussion of pertinent anatomy, classification systems, causes, clinical evaluation and treatment options.
JOSEPH M. SHERRILL: Mallet finger is defined as discontinuity of the terminal digital extensor tendon, resulting in extensor lag at the distal interphalangeal joint with or without compensatory hyperextension deformity at the proximal interphalangeal joint and can be caused by both open or closed trauma. The anatomy of the extensor tendon mechanism of the finger includes the central slip of the extensor digitorum commonis tendon.
JOSEPH M. SHERRILL: The radial and ulnar sagittal bands and metacarpophalangeal joint extensor hood, the dorsal interossei both radial and ulnar, the lumbrical tendon which originates from the flexor digitorum profundus tendon is radially based and inserts into the radial lateral band. Additional components of the extensor tendon anatomy include the radial and ulnar lateral bands, which merge distal to the triangular ligament to form the terminal extensor tendon with lateral contributions from the central slip of the extensor tendon mechanism.
JOSEPH M. SHERRILL: Also noted is the oblique retinacular ligament, which is a static structure which originates from the superflexor tendon sheath and inserts into the terminal tendon. The functional anatomy of the extensor tendon mechanism of finger. The classification of mallet finger includes the following types. Type one, which is a closed injury with or without a small dorsal avulsion fracture.
JOSEPH M. SHERRILL: Type two, which is an open injury or laceration of the terminal extensor tendon. Type three, which is an open injury with loss of skin, subcutaneous tissue coverage and tendon substance and type IV is a mallet fracture. Mallet fracture is further subdivided into groups A, which is a transepiphyseal plate fracture in children. Type B, which is a hyperflexion injury with fracture of the dorsal articular surface of 20% to 50% of the distal phalanx without volar
JOSEPH M. SHERRILL: distal interphalangeal joint subluxation and types C consist of an hyperextension injury with fracture of the dorsal articular surface of the distal phalanx of greater than 50%, and with early or late volar subluxation of the distal interphalangeal joint. And subtype and the subtypes of mallet fracture are illustrated in the following slide.
JOSEPH M. SHERRILL: Treatment of acute mallet fingers. Type one is usually non-operatively or closed consisting of splinting or static immobilization of the distal interphalangeal joint in full extension for six to eight weeks, followed by progressive weaning. It should be noted that both dorsal and volar splinting have similar outcomes and I usually consider providing the patient both a dorsal and a volar or palmar based splint to rotate to avoid skin irritation.
JOSEPH M. SHERRILL: Operative treatment of closed injuries is unusual, but one may wish to consider pinning of the distal interphalangeal joint in special situations, such as in treatment of health care workers requiring frequent hand hygiene. My mallet finger splint weaning protocol is as follows. I basically ask patients to remove the splint for an additional two hours weekly for light activities
JOSEPH M. SHERRILL: as long as no extensor lag develops at the distal interphalangeal joint. If extensor lag develops, the patient is asked to resume continuous or 24 hour per day splinting and to notify the physician. If there is no extensor lag after the four week weaning protocol, the patient is allowed full mobilization. The treatment of acute mallet fingers,
JOSEPH M. SHERRILL: types two and three consist of debridement and tendon repair by suture with pin or splint immobilization. The treatment of acute mallet finger type IV is based on the fracture subtype type IV A or epiphyseal plate. Fractures in children are treated by longitudinal pinning spanning the distal interphalangeal joint with care taken to align the distal phalangeal fracture and to replace the avulsed nail plate beneath the proximal eponychial fold.
JOSEPH M. SHERRILL: Type IV B or hyperflexion injuries with fracture of the dorsal articular surface of the distal phalanx without joint subluxation are treated with closed splinting if no significant volar distal interphalangeal joint subluxation is noted and if reasonable fracture fragment alignment is noted. Type IV C or hyperextension injuries with fracture of the dorsal articular surface greater than 50% associated with early or late volar
JOSEPH M. SHERRILL: subluxation of the distal interphalangeal joint are treated by closed reduction and extension block pinning as is illustrated in the following slide. The treatment of chronic mallet finger includes the following options; distal interphalangeal joint splinting can be considered for delayed presentation of mallet finger for up to three months.
JOSEPH M. SHERRILL: Additional treatment options include extensor tendon repair with or without dermodesis Fowler tenotomy of the central slip of the extensor tendon mechanism, which allows an average of 30 degrees of correction of the extensor lag at the distal interphalangeal joint. Oblique retinacular ligament reconstruction using a lateral band or tendon graft is considered for extensor lag greater than 40 degrees
JOSEPH M. SHERRILL: and in addition, if there is arthritis at the distal interphalangeal joint, arthrodesis is an option. These treatment options are also illustrated in the following slide. The treatment of chronic mallet deformity with associated swan neck deformity is based on the patient's ability to extend the distal interphalangeal joint
JOSEPH M. SHERRILL: if the proximal interphalangeal joint hyperextension laxity is passively blocked. If the patient has the ability to actively extend the distal interphalangeal joint, if the proximal interphalangeal joint is blocked in flexion, the best treatment option is to correct the proximal interphalangeal joint hyperextension laxity by either volar plate repair, flexor digitorum sublimis tenodesis or lateral band translocation as illustrated.
JOSEPH M. SHERRILL: However, if the patient has inability to actively extend the distal interphalangeal joint, if the proximal interphalangeal joint is blocked in flexion, the best treatment options include a Fowler tenotomy if the extensor lag at the distal interphalangeal joint is around 30 degrees and if the proximal interphalangeal joint volar laxity is mild or oblique retinacular ligament can be reconsidered.
JOSEPH M. SHERRILL: Reconstruction can be considered with lateral band or tendon graft if extensor lag at the distal interphalangeal joint is greater than 40 degrees, and if the proximal interphalangeal joint volar laxity is more significant. Swan neck deformity is defined as hyperextension of the proximal interphalangeal joint and concomitant flexion of the distal interphalangeal joint. The causes of Swan neck deformity include extrinsic, intrinsic and articular abnormalities.
JOSEPH M. SHERRILL: Type one or extrinsic causes are due to proximal interphalangeal joint hyperextension caused by excessive traction on the common digital extensor tendon and include disruption of the terminal tendon outside a mallet finger which allows lateral bands to migrate proximally over time. Chronic post-traumatic wrist or metacarpophalangeal joint flexion contractures, tendinous adhesions or muscular shortening of the common digital extensor tendons or extrinsic spasticity of the common digital extensor tendons.
JOSEPH M. SHERRILL: Type two or intrinsic causes, are due to overactivity or contracture of the interosseous or intrinsic muscles and include spasticity of the intrinsics associated with neurological conditions. Fibrosis with ischemic, intrinsic muscle contraction as in compartment syndrome, ulnar intrinsic tendon shortening or contracture in rheumatoid arthritis associated with metacarpophalangeal joint, flexion and volar subluxation and ulnar drift.
JOSEPH M. SHERRILL: And in addition, adhesions or scarring of the intrinsic tendon with contracture. Type three or articular causes of Swan neck deformity are due to weakening or destruction of the volar stabilizing mechanism at the proximal interphalangeal joint and include volar plate disruption, either open or closed, articular fracture with resulting proximal interphalangeal joint volar instability,
JOSEPH M. SHERRILL: loss of flexor digitorum sublimis function including surgical or traumatic disruption or paralysis. Attenuation of volar plate or oblique retinacular ligament secondary to articular synovitis, acquired dorsal proximal interphalangeal joint contracture and congenital proximal interphalangeal joint volar laxity.
JOSEPH M. SHERRILL: The classification of Swan neck deformity includes the following types. Type I includes full range of motion of the digit, no intrinsic tightness and no functional limitations. In type II swan neck deformity, there is intrinsic tightness, as noted, on a Bunnell's test in which
JOSEPH M. SHERRILL: the metacarpophalangeal joint is flexed and the proximal interphalangeal joint can be fully flexed and if the metacarpophalangeal joint is extended, there is limited passive flexion at the proximal interphalangeal joint. Type III includes a stiff proximal interphalangeal joint in all positions of metacarpophalangeal joint, range of motion, but with joint preservation and type IV includes severe arthritic changes of either the proximal interphalangeal joint or distal interphalangeal joint.
JOSEPH M. SHERRILL: The clinical evaluation of Swan neck deformity includes pertinent history and physical exam. History, the history includes discussion of pre-existing congenital laxity, trauma, either laceration or dislocation and inflammatory arthritis. The physical exam includes examination of joint swelling at the proximal or distal interphalangeal joint,
JOSEPH M. SHERRILL: joint range of motion flexor digitorum, sublimis integrity, intrinsic tightness as noted on Bunnell testing and intrinsic spasticity. Clinical evaluation of Swan neck deformity also includes X-ray findings. X-ray may demonstrate fracture of the dorsal proximal aspect of the distal phalanx,
JOSEPH M. SHERRILL: fracture of the volar proximal base of the middle phalanx or arthritis at the proximal or distal interphalangeal joint. Non-surgical management of Swan neck deformity includes observation. Figure 8 or silver ring splinting to block proximal interphalangeal joint hyperextension, but to allow proximal interphalangeal joint flexion.
JOSEPH M. SHERRILL: The surgical treatment of Swan neck deformity is based on previously described types of pathology and includes type I in which there is full range of motion, no intrinsic tightness, no functional limitations. If the patient has the ability to actively extend the distal interphalangeal joint with the proximal interphalangeal joint blocked in flexion, then surgical treatment options include volar plate repair, flexor digitorum sublimis tenodesis, lateral band translocation and Fowler tenotomy for mild proximal interphalangeal joint hyperextension and less than 30 degrees of extensor lag at the distal interphalangeal joint.
JOSEPH M. SHERRILL: However, if the patient has inability to actively extend the distal interphalangeal joint with the proximal interphalangeal joint blocked in flexion and demonstrates greater degrees of proximal interphalangeal joint laxity and greater degrees of extensor lag beyond 40 degrees then surgical considerations would include oblique retinacular ligament reconstruction.
JOSEPH M. SHERRILL: The surgical treatment of Swan neck deformity type II with intrinsic tightness depends on whether the intrinsic tightness is due to contracture or spasticity. If the intrinsic tightness is due to contracture, then surgical treatment consists of intrinsic release or lengthening, and if the tightness is due to spasticity, then the surgical treatment consists of intrinsic tendon release or lengthening or neurectomy of the motor branch of the ulnar nerve
JOSEPH M. SHERRILL: in more severe neurological spasticity. The surgical treatment of Swan neck deformity type three, in which the patient has a stiff proximal interphalangeal joint in all positions of metacarpophalangeal joint, but with joint preservation at the proximal interphalangeal joint can be treated by stepwise procedure as illustrated consisting of proximal interphalangeal joint manipulation or capsular release, skin release, lateral band mobilization with or without translocation or oblique retinacular ligament reconstruction,
JOSEPH M. SHERRILL: central slip lengthening and at the conclusion of the procedure, it's necessary to either check, flex or glide through a small open incision, or if the procedure can be performed under wide awake local anesthesia without tourniquet in which active range of motion in the digit can be assured. The surgical treatment of Swan neck deformity type IV with severe arthritic changes can include proximal interphalangeal joint arthroplasty with soft tissue reconstruction
JOSEPH M. SHERRILL: or proximal or distal interphalangeal joint arthrodesis. Thank you.
Segment:0 .
JOSEPH M. SHERRILL: Right. Tendon Imbalances. Dr. Joseph M Sherrill. This discussion of tendon imbalances focuses on the diagnosis and treatment of mallet finger and swan neck deformity and includes discussion of pertinent anatomy, classification systems, causes, clinical evaluation and treatment options.
JOSEPH M. SHERRILL: Mallet finger is defined as discontinuity of the terminal digital extensor tendon, resulting in extensor lag at the distal interphalangeal joint with or without compensatory hyperextension deformity at the proximal interphalangeal joint and can be caused by both open or closed trauma. The anatomy of the extensor tendon mechanism of the finger includes the central slip of the extensor digitorum commonis tendon.
JOSEPH M. SHERRILL: The radial and ulnar sagittal bands and metacarpophalangeal joint extensor hood, the dorsal interossei both radial and ulnar, the lumbrical tendon which originates from the flexor digitorum profundus tendon is radially based and inserts into the radial lateral band. Additional components of the extensor tendon anatomy include the radial and ulnar lateral bands, which merge distal to the triangular ligament to form the terminal extensor tendon with lateral contributions from the central slip of the extensor tendon mechanism.
JOSEPH M. SHERRILL: Also noted is the oblique retinacular ligament, which is a static structure which originates from the superflexor tendon sheath and inserts into the terminal tendon. The functional anatomy of the extensor tendon mechanism of finger. The classification of mallet finger includes the following types. Type one, which is a closed injury with or without a small dorsal avulsion fracture.
JOSEPH M. SHERRILL: Type two, which is an open injury or laceration of the terminal extensor tendon. Type three, which is an open injury with loss of skin, subcutaneous tissue coverage and tendon substance and type IV is a mallet fracture. Mallet fracture is further subdivided into groups A, which is a transepiphyseal plate fracture in children. Type B, which is a hyperflexion injury with fracture of the dorsal articular surface of 20% to 50% of the distal phalanx without volar
JOSEPH M. SHERRILL: distal interphalangeal joint subluxation and types C consist of an hyperextension injury with fracture of the dorsal articular surface of the distal phalanx of greater than 50%, and with early or late volar subluxation of the distal interphalangeal joint. And subtype and the subtypes of mallet fracture are illustrated in the following slide.
JOSEPH M. SHERRILL: Treatment of acute mallet fingers. Type one is usually non-operatively or closed consisting of splinting or static immobilization of the distal interphalangeal joint in full extension for six to eight weeks, followed by progressive weaning. It should be noted that both dorsal and volar splinting have similar outcomes and I usually consider providing the patient both a dorsal and a volar or palmar based splint to rotate to avoid skin irritation.
JOSEPH M. SHERRILL: Operative treatment of closed injuries is unusual, but one may wish to consider pinning of the distal interphalangeal joint in special situations, such as in treatment of health care workers requiring frequent hand hygiene. My mallet finger splint weaning protocol is as follows. I basically ask patients to remove the splint for an additional two hours weekly for light activities
JOSEPH M. SHERRILL: as long as no extensor lag develops at the distal interphalangeal joint. If extensor lag develops, the patient is asked to resume continuous or 24 hour per day splinting and to notify the physician. If there is no extensor lag after the four week weaning protocol, the patient is allowed full mobilization. The treatment of acute mallet fingers,
JOSEPH M. SHERRILL: types two and three consist of debridement and tendon repair by suture with pin or splint immobilization. The treatment of acute mallet finger type IV is based on the fracture subtype type IV A or epiphyseal plate. Fractures in children are treated by longitudinal pinning spanning the distal interphalangeal joint with care taken to align the distal phalangeal fracture and to replace the avulsed nail plate beneath the proximal eponychial fold.
JOSEPH M. SHERRILL: Type IV B or hyperflexion injuries with fracture of the dorsal articular surface of the distal phalanx without joint subluxation are treated with closed splinting if no significant volar distal interphalangeal joint subluxation is noted and if reasonable fracture fragment alignment is noted. Type IV C or hyperextension injuries with fracture of the dorsal articular surface greater than 50% associated with early or late volar
JOSEPH M. SHERRILL: subluxation of the distal interphalangeal joint are treated by closed reduction and extension block pinning as is illustrated in the following slide. The treatment of chronic mallet finger includes the following options; distal interphalangeal joint splinting can be considered for delayed presentation of mallet finger for up to three months.
JOSEPH M. SHERRILL: Additional treatment options include extensor tendon repair with or without dermodesis Fowler tenotomy of the central slip of the extensor tendon mechanism, which allows an average of 30 degrees of correction of the extensor lag at the distal interphalangeal joint. Oblique retinacular ligament reconstruction using a lateral band or tendon graft is considered for extensor lag greater than 40 degrees
JOSEPH M. SHERRILL: and in addition, if there is arthritis at the distal interphalangeal joint, arthrodesis is an option. These treatment options are also illustrated in the following slide. The treatment of chronic mallet deformity with associated swan neck deformity is based on the patient's ability to extend the distal interphalangeal joint
JOSEPH M. SHERRILL: if the proximal interphalangeal joint hyperextension laxity is passively blocked. If the patient has the ability to actively extend the distal interphalangeal joint, if the proximal interphalangeal joint is blocked in flexion, the best treatment option is to correct the proximal interphalangeal joint hyperextension laxity by either volar plate repair, flexor digitorum sublimis tenodesis or lateral band translocation as illustrated.
JOSEPH M. SHERRILL: However, if the patient has inability to actively extend the distal interphalangeal joint, if the proximal interphalangeal joint is blocked in flexion, the best treatment options include a Fowler tenotomy if the extensor lag at the distal interphalangeal joint is around 30 degrees and if the proximal interphalangeal joint volar laxity is mild or oblique retinacular ligament can be reconsidered.
JOSEPH M. SHERRILL: Reconstruction can be considered with lateral band or tendon graft if extensor lag at the distal interphalangeal joint is greater than 40 degrees, and if the proximal interphalangeal joint volar laxity is more significant. Swan neck deformity is defined as hyperextension of the proximal interphalangeal joint and concomitant flexion of the distal interphalangeal joint. The causes of Swan neck deformity include extrinsic, intrinsic and articular abnormalities.
JOSEPH M. SHERRILL: Type one or extrinsic causes are due to proximal interphalangeal joint hyperextension caused by excessive traction on the common digital extensor tendon and include disruption of the terminal tendon outside a mallet finger which allows lateral bands to migrate proximally over time. Chronic post-traumatic wrist or metacarpophalangeal joint flexion contractures, tendinous adhesions or muscular shortening of the common digital extensor tendons or extrinsic spasticity of the common digital extensor tendons.
JOSEPH M. SHERRILL: Type two or intrinsic causes, are due to overactivity or contracture of the interosseous or intrinsic muscles and include spasticity of the intrinsics associated with neurological conditions. Fibrosis with ischemic, intrinsic muscle contraction as in compartment syndrome, ulnar intrinsic tendon shortening or contracture in rheumatoid arthritis associated with metacarpophalangeal joint, flexion and volar subluxation and ulnar drift.
JOSEPH M. SHERRILL: And in addition, adhesions or scarring of the intrinsic tendon with contracture. Type three or articular causes of Swan neck deformity are due to weakening or destruction of the volar stabilizing mechanism at the proximal interphalangeal joint and include volar plate disruption, either open or closed, articular fracture with resulting proximal interphalangeal joint volar instability,
JOSEPH M. SHERRILL: loss of flexor digitorum sublimis function including surgical or traumatic disruption or paralysis. Attenuation of volar plate or oblique retinacular ligament secondary to articular synovitis, acquired dorsal proximal interphalangeal joint contracture and congenital proximal interphalangeal joint volar laxity.
JOSEPH M. SHERRILL: The classification of Swan neck deformity includes the following types. Type I includes full range of motion of the digit, no intrinsic tightness and no functional limitations. In type II swan neck deformity, there is intrinsic tightness, as noted, on a Bunnell's test in which
JOSEPH M. SHERRILL: the metacarpophalangeal joint is flexed and the proximal interphalangeal joint can be fully flexed and if the metacarpophalangeal joint is extended, there is limited passive flexion at the proximal interphalangeal joint. Type III includes a stiff proximal interphalangeal joint in all positions of metacarpophalangeal joint, range of motion, but with joint preservation and type IV includes severe arthritic changes of either the proximal interphalangeal joint or distal interphalangeal joint.
JOSEPH M. SHERRILL: The clinical evaluation of Swan neck deformity includes pertinent history and physical exam. History, the history includes discussion of pre-existing congenital laxity, trauma, either laceration or dislocation and inflammatory arthritis. The physical exam includes examination of joint swelling at the proximal or distal interphalangeal joint,
JOSEPH M. SHERRILL: joint range of motion flexor digitorum, sublimis integrity, intrinsic tightness as noted on Bunnell testing and intrinsic spasticity. Clinical evaluation of Swan neck deformity also includes X-ray findings. X-ray may demonstrate fracture of the dorsal proximal aspect of the distal phalanx,
JOSEPH M. SHERRILL: fracture of the volar proximal base of the middle phalanx or arthritis at the proximal or distal interphalangeal joint. Non-surgical management of Swan neck deformity includes observation. Figure 8 or silver ring splinting to block proximal interphalangeal joint hyperextension, but to allow proximal interphalangeal joint flexion.
JOSEPH M. SHERRILL: The surgical treatment of Swan neck deformity is based on previously described types of pathology and includes type I in which there is full range of motion, no intrinsic tightness, no functional limitations. If the patient has the ability to actively extend the distal interphalangeal joint with the proximal interphalangeal joint blocked in flexion, then surgical treatment options include volar plate repair, flexor digitorum sublimis tenodesis, lateral band translocation and Fowler tenotomy for mild proximal interphalangeal joint hyperextension and less than 30 degrees of extensor lag at the distal interphalangeal joint.
JOSEPH M. SHERRILL: However, if the patient has inability to actively extend the distal interphalangeal joint with the proximal interphalangeal joint blocked in flexion and demonstrates greater degrees of proximal interphalangeal joint laxity and greater degrees of extensor lag beyond 40 degrees then surgical considerations would include oblique retinacular ligament reconstruction.
JOSEPH M. SHERRILL: The surgical treatment of Swan neck deformity type II with intrinsic tightness depends on whether the intrinsic tightness is due to contracture or spasticity. If the intrinsic tightness is due to contracture, then surgical treatment consists of intrinsic release or lengthening, and if the tightness is due to spasticity, then the surgical treatment consists of intrinsic tendon release or lengthening or neurectomy of the motor branch of the ulnar nerve
JOSEPH M. SHERRILL: in more severe neurological spasticity. The surgical treatment of Swan neck deformity type three, in which the patient has a stiff proximal interphalangeal joint in all positions of metacarpophalangeal joint, but with joint preservation at the proximal interphalangeal joint can be treated by stepwise procedure as illustrated consisting of proximal interphalangeal joint manipulation or capsular release, skin release, lateral band mobilization with or without translocation or oblique retinacular ligament reconstruction,
JOSEPH M. SHERRILL: central slip lengthening and at the conclusion of the procedure, it's necessary to either check, flex or glide through a small open incision, or if the procedure can be performed under wide awake local anesthesia without tourniquet in which active range of motion in the digit can be assured. The surgical treatment of Swan neck deformity type IV with severe arthritic changes can include proximal interphalangeal joint arthroplasty with soft tissue reconstruction
JOSEPH M. SHERRILL: or proximal or distal interphalangeal joint arthrodesis. Thank you.
Form Title Download
Transcript
Form Title Bookmark
Entire Media
Segment(s)
Custom Clip
Start At:
End At:
Form Title Share
Entire Media
Segment(s)
Custom Clip
Start At:
End At:
Form Title Accessibility and Keyboard Shortcuts
The cadmore media player is a professional video and audio player experience designed to deliver media as well as relevant transcript, segmentation, and metadata. On the screen is a traditional html video player with buttons such as play, pause, forward, captioning, language and play speed selection and more. Adjacent to the video player are generally tabs, one of which shows a scrolling transcript of the video and the other shows any segmentation of the media, sometimes referred to as chapters. At the top of the video is a menu bar containing an Explore button and Tools button. The Explore button allows you to dig deeper into the video by opening up an explore dialog which contains tabs for a visual navigation of the video using thumbnails, an about tab which contains rich metadata about the media, a segments tab which contains further information about the video, and a related tab which contains links to other media related to this media. The tools button opens a menu with options for sharing the media on social media, creating bookmarking links, creating embed codes for your website, generating citations, and more.
Keyboard Shortcuts
Keyboard shortcuts only work while you are in forms mode. By entering forms mode, you will be able to quickly access portions of the media player with single key commands.
Spacebar: Play/Pause Toggle
I: Info Menu,
O: Tools Menu
T: Transcript Tab,
S: Segments Tab
F: Forward 15,
R: Rewind 15
L: Languages,
P: Playspeed
M: Mute Toggle,
V: Volume Bar
N: Video Only Toggle,
B: Full Screen Toggle
C: Captions Toggle
Escape: Closes dialogs and menus / Exits Player
Z: Next Transcript Chapter
X: Previous Transcript Chapter
Q: Next Transcript Paragraph
W: Previous Transcript Paragraph
Entire Media
Segment(s)
Custom Clip
Start At:
End At:
Embed Size
-
You Size - Responsive
-
We Size - Responsive
-
Picture Overlay Popup
-
400 x 225
-
512 x 288
-
400 x 700
-
768 x 1024
-
1024 x 512
-
1280 x 608
Form Title Cite
No citation provided.
Info
Segments
Related
Thumbnails
Exit Clip Mode
You have requested to see a portion of the video outside of the clip.
Click OK to switch to the full video or click if you would like to stay inside the clip. You can always go back to the clip with Tools and Clip Mode